Stay Away From Low Carb Diets To Gain Muscle Weight

Stay Away From Low Carb Diets To Gain Muscle Weight

Copyright 2006 Jonathan Perez

Many people looking to gain muscle weight can become very confused when they read and hear all of the conflicting information that's spread on TV, magazines, the internet, and diet books.

One of the most misunderstood techniques is the low carb diets that have been very popular in the last several years.

The popular belief lately is that sugar (carbohydrates) should be kept to moderate to low levels in your eating, even if you are trying to build muscle mass and / or gain muscle weight.

The reason for this is because of the recent popularity of low-carb diets, such as The Atkins diet, The Southbeach diet, The Zone diet, etc.

Even in bodybuilding magazines all of a sudden everyone and their mother is recommending to keep carbs in your diets almost to a minimum.

Well, the misconception that sugar is somehow "evil" and will cause you to get fat, raise your bad cholesterol, and even lose your "insulin sensitivity" is based on several MYTHS that have been passed around by the media and "monkey see, monkey do" nutrition and exercise "gurus".

One of the main misconceptions is that eating carbs will cause a sharp rise of insulin in your blood, which supposedly causes your body to store fat.

A lot of this is based on the incorrect belief that you should avoid eating "high GI (glycemic index)" foods, and to mainly get most of your carbs from "low GI" foods.

Basically, the Glycemic Index is a chart that was developed years ago that measures how much of a rise in insulin a certain food causes in the blood once digested.

To put it simply, the higher the GI number is, the higher and quicker it makes your insulin rise.

(For some odd reason that's NOT based on any real, conclusive evidence. People seem to think that raising your insulin levels is "bad").

Well, in 1981, researchers at the University of Toronto were the first to accurately notice that "simple" carb foods (having a high GI) actually produced a smaller increase in blood sugar than most "complex" (or low GI) foods (everyone today thinks the opposite is true, of course based on what they read in the latest "how to gain muscle weight" article)!

In fact, eating table sugar (sucrose) actually causes a smaller increase in blood sugar than eating foods such as baked potatoes and whole wheat bread.

Foods containing added sugars do NOT have a higher GI than foods containing naturally occurring sugars.

In fact, you really can't count on the "GI" much at all!!!

Why?

Because the Glycemic Index does NOT take into account the differences in variety, ripeness, processing, or cooking foods, which have a huge impact on how that particular food affects your insulin levels, even if you are trying to gain muscle weight.

Also, the fat content of food also has an affect on insulin: foods that are high in fat have a lower GI, like peanuts, which only have a GI of 14.

In addition, there some foods that have a high GI number, but don't affect your insulin levels at all........like carrots!

Carrots have a GI of 95 (which is pretty high), but don't try to tell me that you'll get fat, get diabetes, etc., from eating carrots!!!

Bodybuilding magazines, supplement companies, and the internet spread myths to all of us seeking to build muscle mass, only leading us to NO RESULTS!

I guarantee you that if you are desperately trying to gain muscle, if you go on a low carb diet, not only are you not going to build any muscle weight, but you are actually going to get smaller than what you are already are.

If you are trying to eat your way to a leaner, more defined, more sculpted you, there is NO NEED TO BREAK DOWN YOUR EATING DOWN TO ALL KINDS OF RATIOS or PERCENTAGES.

Regardless of what type of foods you eat and in what combinations, "IT ALL COMES DOWN TO THE CALORIES"!

BONUS : Steroid Articles And Information And Their Side Effects - Shrenksonlinepharma

Contrary to what many would expect, this compound is actually only a weak agonist of the androgen receptor (AR), with poor binding. It follows, then, that its value must mostly come from non-AR-mediated effects. It is therefore a Class II steroid. Since it is not very effective in activating ARs, it should be stacked with a Class I steroid that is effective in
this regard, such as Primobolan, Deca Durabolin, or trenbolone acetate. There is no point in stacking it with Anadrol®, which has similar activity -- one ought to simply use the more appropriate drug. With testosterone or Deca, Dianabol is to be preferred; with Primobolan or trenbolone acetate, Anadrol® is to be preferred (though Dianabol is still a good choice) because Anadrol® does not aromatize. For an oral-only cycle -- something I don't recommend -- Anadrol® is the better choice in my opinion for that also, at 150 mg/day (preferably divided to 3 or 6 doses.)

Methandrostenolone converts to estradiol via aromatase. The amount of this conversion may be reduced by use of Arimidex, or less preferably Cytadren (see previous articles discussing dosage and dose pattern.) Or if the conversion is allowed, Clomid may be used to block adverse estrogenic effects.

Irreversible hoarsening of the voice has been seen in some women from very few tablets of Dianabol: one per day for a few weeks. For this reason, in the 1960s doctors decided to end what had been a fairly common practice of prescribing this drug at one tab per day to women as a "tonic." It is not a good choice for the woman who chooses to use anabolic steroids.

The usual dosing for men is 25-50 mg/day in divided doses, preferably four or five doses. The drug is 17-alkylated and so use should be limited to no more than 6 weeks, and preferably no more than four weeks, with at least an equal amount of time off.

Trivial name Methandrostenolone
Systematic name 17â-hydroxy-17á-
methyl-1,4-androstadien-3-one
CAS registry number 72-63-9
ATC code A14AA03
Merck Index Number 5978
Chemical formula C20H28O2
Molecular weight 300.435 g/mol
Bioavailability
Metabolism Hepatic
Elimination half-life 6 hours
Excretion Urinary:
Pregnancy category X
Routes of administration Oral